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Exploring the Potential of LSD for Treating Anxiety Disorders in 2026

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LSD and its effects on brain rewiring

LSD May Enhance Brain Rewiring and Alleviate Anxiety

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Two significant trials examining the potential of the hallucinogen LSD to alleviate anxiety are set to conclude in 2026. Early-stage trials have shown promising results, potentially allowing the treatment to be available in the United States by 2027.

Generalized anxiety disorder is prevalent, causing intense anxiety about various issues. Traditional treatments often include antidepressants and talk therapy, yet approximately half of patients do not respond adequately.

Other psychedelics, like psilocybin and MDMA, are already being utilized in certain nations, including Australia and Switzerland, to treat severe depression and PTSD. LSD is gaining attention in mental health studies, primarily because it has been shown to induce deep emotional experiences and may enhance the brain’s capacity to form new neural connections.

A pivotal trial in 2025 demonstrated that a single high dose of LSD significantly reduced moderate to severe anxiety for a duration of at least three months.

Currently, two late-stage trials are ongoing, with results expected in 2026. Each trial involves about 200 participants with moderate to severe anxiety who will receive either 100 micrograms of LSD or a placebo. Their anxiety levels will be monitored over three months post-administration.

In the subsequent 10-month phase, all participants, including those initially on a placebo, will have the opportunity to take LSD if they report their anxiety reaching a predefined threshold on a standardized scale. This design aims to assess the duration of effects following a single dose. Dan Carlin, affiliated with New York biotech firm MindMed, has been involved in both the 2025 trial and the current studies.

The primary distinction between the two studies is that, in the second trial, an additional group will receive 50 micrograms of LSD. The 2025 trial indicated this dosage could induce hallucinations but didn’t effectively diminish anxiety compared to the placebo, thereby addressing a common challenge in psychedelic research—participants often struggle to identify if they received the active drug.

Participants in this third group will be aware they received LSD but will remain uncertain if the dosage is sufficient to alleviate anxiety, as noted by Sandeep Nayak of Johns Hopkins Bayview Medical Center, who is not part of these trials.

If the results of both trials are favorable, the FDA could approve LSD for anxiety treatment in the United States by 2027, potentially leading to approval in Europe and other regions.

“If the upcoming trial confirms results similar to the previous ones from 2025, that should satisfy the FDA,” said Boris Heifetz of Stanford University.

A meaningful impact on patients’ lives is typically defined as a 3 to 5-point difference on the anxiety scale between placebo and LSD groups, as noted by Nayak. The 2025 trial showed a difference of approximately five points, indicating a strong possibility the next trial will meet this threshold. However, any therapeutic benefits must be weighed against the duration of effects and potential side effects.

For instance, temporary psychological distress during treatment might be acceptable to the FDA, unlike long-lasting distress, Nayak stated. Notably, long-term distress was not observed in the 2025 trial.

Even with potential approval, Nayak emphasizes it may take several years for LSD to become widely accessible for anxiety disorders, and it would likely be a last-resort treatment after conventional therapies fail. This is primarily due to the logistical challenges, such as the need for clinicians to supervise patients during psychedelic experiences.

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Source: www.newscientist.com

Antibiotics Generally Do Not Raise the Risk of Autoimmune Disorders

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Antibiotic use may impact the immune system adversely

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A comprehensive study involving over 6 million children reveals that those exposed to antibiotics prenatally or in early childhood do not generally face a heightened risk of developing autoimmune disorders during adolescence. However, the dynamics are quite intricate.

The notion that antibiotics may lead to autoimmunity dates back to the 1980s. David Strachan later proposed that the London’s Faculty of Hygiene and Tropical Medicine illustrates fewer infections in cleaner environments leading to a higher rate of childhood allergies.

This line of thought gave rise to the hygiene hypothesis, suggesting that limited early exposure to specific microorganisms fails to adequately train the immune system, which may react excessively to benign substances, leading to allergic responses or autoimmune disorders. Conditions like type 1 diabetes, inflammatory bowel disease, and lupus occur when immune cells mistakenly target the body’s own tissues.

Numerous studies since have demonstrated the crucial role of various microorganisms, particularly gut microbes, in shaping our immune responses. For instance, essential compounds for the maturation of specialized immune cells, such as regulatory T cells, play pivotal roles in preventing autoimmunity. This raises concerns over whether antibiotics that disrupt gut microbiota could facilitate the emergence of autoimmune diseases.

“Over time, numerous clinical studies, primarily using animal models, have substantiated the idea that antibiotics, or modifications to the gut microbiome, significantly affect immunity,” states Martin Kriegel from the University of Munster, Germany.

For instance, a 2016 study on mice illustrated that repeated antibiotic treatment at early life stages increased susceptibility to type 1 diabetes. Mice with genetic predisposition to this disorder were given their mothers’ breast milk alongside antibiotics thrice at 4 and 5 weeks old. Approximately 50% of the male and 80% of the female mice in this group developed type 1 diabetes by 30 weeks, while only about 25% of the male mice and 50% of the female mice that avoided antibiotics faced the same issue.

Similar correlations have surfaced in human research. An evaluation of over 10 million individuals released this year indicates that those prescribed antibiotics face an 40% higher risk of later developing inflammatory bowel disease. Additionally, a 2019 study involving over 110,000 participants linked antibiotic prescriptions to a 60% increased risk of developing rheumatic arthritis.

Conversely, other studies denote contrary findings. For example, a 2017 study involving over 15,000 children assessed for type 1 diabetes and celiac disease found no correlation between these conditions and antibiotic use prior to the age of four.

Recently, Eun-Young Choi from Sungkyunkwan University in Korea and her colleagues tracked the onset of six autoimmune conditions: type 1 diabetes, chronic pediatric arthritis, ulcerative colitis, Crohn’s disease, lupus, and Hashimoto’s disease. Around 1.5 million mothers received antibiotics during pregnancy, and a second group of 3.4 million children received similar treatment within the first six months post-birth, with 1.9 million treated with antibiotics.

After adjusting for variables like infection type, socioeconomic status, and gender, researchers found no collective correlation between antibiotic exposure in utero or early childhood and the likelihood of developing autoimmune conditions in adolescence.

Why do these findings differ so greatly? The gut microbiota’s complexity plays a significant role. Various factors influence it, making holistic explanations challenging. For instance, the studies referenced may fail to account for dietary influences on gut microbiota.

Different antibiotics also provoke varied effects. Choi’s study established a connection between broad-spectrum antibiotic use during pregnancy and subsequent development of Crohn’s disease in children. Additionally, the timing of antibiotic exposure appeared critical; antibiotics administered within two months of birth correlated with a 30% increased risk of Hashimoto’s disease.

This doesn’t imply that antibiotics should be entirely avoided. “When antibiotics are deemed necessary during pregnancy, it’s due to a belief that their benefits surpass potential risks,” asserts Christopher Zahn from the American University of Obstetricians in Washington, DC. For example, urinary tract infections can result in severe issues like preterm birth and low birth weight.

In fact, certain antibiotics may thwart autoimmunity. A 2018 study discovered that infections from pathogenic bacteria, notably Enterococcus gallinarum, induced autoimmunity in mice. Antibiotic treatment not only prevented mortality but also inhibited immune cells from attacking the organisms’ tissues.

“Thus, the situation is immensely complicated,” remarks Kriegel.

However, the latest findings should alleviate concerns for pregnant individuals and those with young children, reassures Zahn.

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Source: www.newscientist.com

Neurological Disorders May Be Linked to Neanderthal DNA

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The intersection of anatomically modern Homo sapiens and Neanderthals thousands of years ago might contribute to Chiari malformation type 1, a serious neurological condition that affects an estimated 1% of today’s population.

Homo sapiens: Homo erectus, Homo heidelbergensis, and Homo neanderthalensis. Plomp et al. utilized 3D data and geometric morphometry to investigate this hypothesis. Image credit: Neanderthal Museum.” width=”580″ height=”618″ srcset=”https://cdn.sci.news/images/2014/04/image_1837-Neanderthal.jpg 580w, https://cdn.sci.news/images/2014/04/image_1837-Neanderthal-281×300.jpg 281w” sizes=”(max-width: 580px) 100vw, 580px”/>

In 2013, scientists surmised that Chiari malformation type 1 might arise from cranial development genes tracing back to three smaller extinct Homo species: Homo erectus, Homo heidelbergensis, and Homo neanderthalensis. Plomp et al. employed 3D data and geometric morphometry to evaluate this hypothesis. Image credit: Neanderthal Museum.

Chiari malformation type 1 occurs when the rear of the skull is insufficiently sized to accommodate the brain, causing a portion of the brain to protrude from the skull into the spinal canal.

This condition can compress the herniated brain tissue, resulting in symptoms such as headaches, neck pain, and dizziness. In severe instances, this can lead to life-threatening complications.

“In medicine, as in all sciences, clarifying the causal chain is vital.”

“The clearer the causal relationships, the greater the chances that the condition can be effectively managed or even resolved.”

“Further testing of the hypothesis is needed, but our study may bring us a step closer to understanding the causal chain that leads to Chiari malformation type 1.”

In 2010, genetic evidence emerged indicating that modern humans interbred with Neanderthals tens of thousands of years ago.

Individuals of non-African descent carry approximately 2-5% Neanderthal DNA, tracing back to these ancestral mating interactions.

The hypothesis suggesting that Chiari malformation type 1 may stem from ancient Homo genes entering the human gene pool through mating was first proposed by researcher Evens Barbosa Fernandez from the University of Campinas.

Dr. Fernandez hypothesized that the differences in skull structure between modern humans and other Homo species play a significant role in causing malformations.

In this study, Professor Mark Collado, PhD, Kimberly Promp at the University of the Philippines Diliman, and colleagues employed modern imaging techniques and advanced statistical shape analysis to compare 3D models of living humans with those of Homo sapiens, Neanderthals, Homo heidelbergensis, and Homo erectus.

The findings revealed that individuals with Chiari malformations exhibit more similar skull shapes to Neanderthals than those without such malformations.

Notably, all other fossil skulls align more closely with modern humans lacking Chiari malformation type 1, supporting the hypothesis that certain individuals today carry Neanderthal genes that influence skull shape, potentially leading to a mismatch between skull dimensions and the shape of the modern human brain.

This discrepancy could result in insufficient space within the skull, causing the brain to extend into the spinal cord, which is the only available outlet.

Given that various global populations possess different levels of Neanderthal DNA, this study suggests that certain European and Asian populations may be more susceptible to Chiari malformation type 1, although additional research is needed to validate this.

“The study of archaeology and human evolution is not merely fascinating,” Professor Collado remarked.

“It could offer insights into current health challenges.”

“In this instance, fossils have provided clarity about the condition, but numerous modern issues also help illuminate archaeological and paleontological findings.”

Study published in the journal Evolution, Medicine, Public Health.

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Kimberly Promp et al. 2025. An ancient hypothesis regarding Homo introduction and Chiari malformation type I Evolution, Medicine, Public Health 13(1):154-166; doi:10.1093/end/eoaf009

Source: www.sci.news

Babies Created with Three DNA Sources Are Free from Genetic Disorders

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The baby became pregnant via IVF

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Eight young children seem to be shielded from severe genetic disorders following their birth through a three-parent DNA technique. This method involved replacing defective maternal mitochondria with those from a female donor.

Approximately 1 in 5,000 individuals carry mitochondrial defects that provide energy to cells and are inherited solely from the mother. Such defects may result from genetic mutations leading to issues like blindness, seizures, and, in extreme cases, death. “Families find it incredibly challenging to cope with these diseases. They are heart-wrenching,” states Bobby McFarland from Newcastle University, UK.

In 2015, the UK first sanctioned a procedure called pronuclear transfer for women at high risk of passing on mitochondrial conditions, particularly those who cannot benefit from pre-implantation genetic testing.

This nuclear transfer technique utilizes eggs from both mothers and donors, which are fertilized with paternal sperm via IVF. After roughly 10 hours, the nuclei from both eggs are extracted, leaving behind the crucial genetic material that is separate from mitochondrial DNA.

The nucleus from the mother is subsequently inserted into the donor’s egg, yielding an embryo that primarily inherits DNA from its biological parent while acquiring mitochondria from the donor. Some mitochondrial DNA from the mother may still be unintentionally transmitted, according to Burt Smeet from Maastricht University in the Netherlands.

McFarland, who pioneered this method with her team, has applied the technique to 19 women harboring harmful mutations in over 80% of their mitochondria, typically the level that causes issues.

Seven of the women achieved pregnancy after the final embryo transfer, resulting in eight healthy births, including one pair of twins.

The researchers evaluated blood samples from the newborns, finding no harmful mitochondrial DNA mutations in five, and only trace levels in the remaining three. “The results have exceeded expectations,” says Mike Murphy from Cambridge University.

In the months or years following these nuclear transfers, all children have shown progress with developmental milestones. However, some may encounter complications that may or may not be linked to the procedure. For instance, one child developed high blood fat levels and an abnormal heartbeat, both of which were successfully addressed, while another experienced epilepsy at 7 months old, which resolved on its own.

The research team plans to monitor these children to assess the long-term consequences of the procedure.

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Source: www.newscientist.com

Brain Changes from Eating Disorders Mirror Those Seen in OCD and Autism

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False-colored nuclear magnetic resonance images of children’s brains

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New research indicates that children with anorexia nervosa are undergoing significant brain changes that go beyond what starvation can explain. This insight helps clarify the neurological mechanisms behind the disorder, potentially paving the way for improved treatment strategies.

Anorexia nervosa is noted for severe dietary restrictions and a distorted body image, making it a less understood condition. While previous studies have highlighted that the brain’s outer layer, or cortex, is notably thinner in these individuals, it remains uncertain whether such changes stem from malnutrition or are intrinsic to anorexia.

Clara Morrow from The University of Montreal, Canada, examined brain scans of children with anorexia alongside those with Avoidant/Restrictive Food Intake Disorder (ARFID). Although both conditions encompass significant food restrictions and weight loss, ARFID lacks the body image concerns that characterize anorexia. Instead, individuals with ARFID may avoid food due to sensory sensitivities, disinterest in eating, or fear of adverse consequences like choking, vomiting, or gastrointestinal distress. The comparison could shed light on the unique brain changes associated with each condition and malnutrition, according to Moreau.

The study analyzed brain scans from 124 children diagnosed with anorexia, 50 with ARFID, and 116 without eating disorders. All participants were under 13 years old and resided in France. Researchers examined the extent of brain differences between those with and without eating disorders.

On average, children diagnosed with anorexia exhibited a significantly thinner cortex compared to those without eating disorders. Once body mass index (BMI) was taken into account, anorexia correlated with cortical thinning across 32 brain regions, particularly in the superior head lobule, an area involved in sensory information processing. “This aligns with our understanding, as we know anorexic patients often struggle with their perception of weight and size,” stated team member Anael Ayrolles from the University of Paris.


These alterations are akin to those observed in older adolescents and adults suffering from anorexia, notes Moreau. “The effect size is among the most significant in psychiatry,” she comments. “It appears as if they’ve experienced accelerated brain aging or early Alzheimer’s disease, though they show no symptoms of Alzheimer’s. However, if their BMI is normalized, brain recovery is often observed, though not in every case.”

In contrast, no significant differences in cortical thickness were observed between children with ARFID and those without any eating disorders. “We anticipated some overlap with anorexia potentially reflective of BMI,” explains Moreau. “However, our findings did not reveal many similarities between the two conditions.” The reason for this remains unclear, especially since this is the inaugural brain imaging study focused on ARFID. Given that ARFID typically manifests before the age of five, the brain may have adapted to limited food intake, suggests Moreau.

The researchers subsequently contrasted these brain differences with findings from previous studies on other disorders, including obsessive-compulsive disorder (OCD), ADHD, and autism. They found a notable correlation between anorexia and OCD, whereas ARFID displayed brain changes similar to those associated with autism. This aligns with Moreau’s assertion that sensory sensitivity is prevalent in both autism and ARFID. Conversely, OCD and anorexia exhibit obsessions, rituals, and preconceived notions.

Nevertheless, individuals with OCD and anorexia frequently present other mental health challenges, notes Joanna Steinglass from Columbia University in New York. Approximately 14% of those diagnosed with anorexia also meet the criteria for OCD. This complicates the understanding of whether a genuine neurological resemblance exists between the two conditions or if other mental health challenges underpin this correlation.

“We were cautious not to over-interpret our results,” said Ayrolles. However, these discoveries imply that malnutrition alone may not account for all the brain changes observed in anorexia. “Mental illness is fundamentally a brain-based illness, and understanding this helps us address patient experiences more effectively, often leading to less blame,” remarks Steinglass. “This insight could drive the development of more effective treatments.”

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Source: www.newscientist.com

Life-Saving Treatments for Fatal Genetic Disorders Through Brain Immune Cell Replacement

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Microglia are specialized immune cells in the brain

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The process of replacing immune cells in the brain halts the advancement of a rare and terminal brain disorder known as ALSP. This also paves the way for future clinical trials targeting other neurological ailments.

Extensive research indicates that impaired microglia—specialized immune cells within the brain—play a role in various neurological disorders, including Alzheimer’s disease and schizophrenia. The term ALSP stands for adult-onset leukoencephalopathy with axonal spheroids and pigmented glia, characterized by mutations in genes responsible for the survival of these cells, resulting in a reduced number of microglia and leading to progressive cognitive decline. Currently, no effective treatment exists for this fatal illness.

To address this, Bo Peng from Fudan University in China and his team employed a novel treatment called microglia replacement therapy. Prior experiments in rodents have shown that implanted stem cells—capable of developing into different cell types—can effectively replace microglia. However, it is necessary to first eliminate existing microglia in the brain to facilitate this. This can be achieved using drugs that target protein microglia.

Pursuing this avenue, Peng and his colleagues conducted initial tests on five mice with genetic mutations analogous to those associated with ALSP. As the mutations already impacted protein microglia, the researchers did not need to deplete these proteins with medication. Subsequently, they transplanted stem cells from healthy mice into the affected mice. Fourteen months later, treated mice exhibited approximately 85% more microglia in their brains compared to six untreated mice harboring the same mutation. Notably, these treated mice also demonstrated improvements in motor function and memory.

Encouraged by these promising findings, the researchers extended the treatment to eight individuals diagnosed with ALSP, using donor stem cells without preconditions. One year post-treatment, brain scans revealed minimal changes in participants compared to scans taken before the procedure. In contrast, four untreated individuals displayed significant brain deterioration and lesions over the same period. This implies that microglial replacement therapy effectively halted the progression of the disease.

At the study’s outset, all participants underwent cognitive assessments using a 30-point scale, where a decrease in score indicated cognitive decline. Reassessments a year later showed that, on average, scores remained stable for those who received the microglia replacements.

These results point to microglial replacement therapy being a potentially effective solution for ALSP. However, since this represents the inaugural human trial, “we remain unaware of any potential side effects,” comments Peng. “Given the rapidly progressive and lethal nature of this disease, prioritizing benefits over possible side effects might be crucial.”

Chris Bennett from the University of Pennsylvania cites the historical use of stem cell transplants for treating neurological disorders. “It has demonstrated effectiveness, particularly through microglia replacement,” he states. Recent FDA approvals for two similar therapies addressing other rare brain conditions further support this. “While prior studies may not have used this exact terminology, they effectively addressed similar conditions,” Bennett elaborates. “I’d describe this as a smart and innovative application of stem cell transplants. Nonetheless, microglia replacement therapy has been evolving for decades.”

Despite this, the results underscore the broader implications of microglial replacement therapy. Experts believe this strategy could one day address more prevalent brain disorders. For example, certain genetic mutations significantly heighten Alzheimer’s disease risk and affect microglial function. Replacing these malfunctioning cells with healthy human equivalents could offer a promising avenue for treatment.

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  • Neuroscience /
  • Immune System

Source: www.newscientist.com

Different Forms and Varieties of Eating Disorders

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Eating Disorders: The Basics debunks the stereotype that eating disorders only affect a certain group of people and emphasizes their universal impact and the need for comprehensive, early treatment and support. I am.

According to eating disorder experts, eating disorders affect everyone, regardless of race, gender, or age.

Medical experts say the myth that only thin, white, wealthy girls develop eating disorders is preventing other patients from receiving diagnosis and treatment.

Psychiatrist Janet Treasure, general practitioner Dr Elizabeth McNaught and therapist Jess Griffiths (all survivors of eating disorders) agree that this stereotype is not true for other people, including black women and men. She says it means she has a hard time asking for help.

They urge clinicians to treat all eating disorders as serious, even those that don’t involve weight loss, such as purging.

The importance of early intervention and comprehensiveness in treatment

Professor Treasure, Dr McNaught and Jess from King’s College London also highlight the importance of early intervention to save lives and the important role of fathers in helping girls recover.

their books Eating disorders: the basics – TV presenter Strictly Winner Stacey Dooley – For schools, health care workers, and families.

This guide details common risk factors, different types of eating disorders, the latest treatments, and provides advice for families on how to support their loved one’s recovery.

“Eating disorders are often thought of as affecting thin, white, affluent girls. But they lack true discrimination about who they affect,” the authors write. states.

“Other groups, such as men, racial minorities, transgender people, and people from disadvantaged socio-economic backgrounds, can remain in our communities struggling with eating disorders for years untreated. There is a gender.

“It is also important to recognize that all eating disorders are serious and all eating disorders deserve treatment and support.

“It doesn’t have to be a lifelong or fatal disease, but it can often be due to lack of preparedness and lack of awareness of symptoms in people who are underweight.”

Recognize the universal impact of eating disorders

Problematic behaviors related to eating food are common around the world. These can occur at any stage of life and affect everyone, regardless of race, gender, or age.

eating disorder Based on the latest evidence on anorexia, bulimia, bulimia, and other conditions.

The guide also features real-life stories from people who have developed eating disorders, as well as letters of hope to support those still suffering.

These stories include those of patients who were told they were not sick enough to need help.

Despite the risks and pain associated with her condition, Carla Lisette said her purging disorder would only be taken seriously if it met the criteria for anorexia.

Furthermore, she added: “Although most people with purge disorder do not become underweight, that does not mean they are at a healthy weight for their bodies or that they are not harming themselves. It doesn’t even mean it. The purge could be deadly.”

In the letter, Christina Taylor was told she was “too healthy” to be helped, even though she drank excessively and felt sick 10 times a day.

“This (receiving the letter) was one of the most invalidating experiences of my life. I truly felt that there was no point in continuing this way.”

Professor Treasure and his co-authors say other challenges remain when it comes to eating disorders, including:

  • Food poverty, ultra-processed foods, and less sharing of home-cooked meals. These are among the environmental factors behind eating disorders.
  • Men often face societal pressure to “be manly.” This can create further secrecy about one’s disorder and create barriers to receiving treatment.
  • Body mass index (BMI) may not be useful in many situations. The authors state that the risk of poor health is related to the degree of weight loss, not absolute body weight. Some people may have a “normal” BMI but still be at risk for serious physical harm.
  • Fathers and partners may feel left out, as if eating disorders are “women’s work”, or siblings may be seen as too young to be involved. However, the authors say it plays an important role in supporting your loved one’s recovery.

References: Eating Disorders: The Basics, by Elizabeth McNaught, Janet Treasure, and Jess Griffiths.
DOI: 10.4324/9781003342762

Source: scitechdaily.com

Unveiling the Reality of Sleep Disorders: When a Night Shift Becomes a Nightmare

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A new study investigated the relationship between shift work patterns, sociodemographic factors, and sleep disorders. They found that shift work, especially night shifts, significantly disrupted sleep, with about a third of all participants reporting at least one sleep disorder. The study also found that demographic factors such as gender, age, and education level influence sleep health.

A new study shows that working night shifts increases the incidence of sleep disorders, especially in young people with low levels of education.

Sleep is important not only for physical and mental health, but also for daytime and neurocognitive function. When people work in shifts (21% of workers in the European Union worked shifts in 2015), their circadian sleep-wake rhythms are often disrupted. Now, Dutch researchers have investigated the relationship between different shift work patterns, sociodemographic factors, and sleep disorders.

“Compared to working regular shifts during the day, working other shift types has been shown to have a higher incidence of sleep disturbances, especially those working rotational or regular night shifts,” GGZ Drenthe said Dr. Marike Lancel, a state mental health researcher.Institute and lead author of the study published in frontiers of psychiatry. “Notably, 51% of those working night shifts tested positive for at least one sleep disorder.”

ask about sleep

“There is a lot of evidence that shift work reduces sleep quality. However, there is little evidence of the impact that different types of shifts have on the prevalence of different sleep disorders and how this varies depending on demographic characteristics. “We know very little about whether they will,” Lancel continued.

To fill these gaps, researchers recruited more than 37,000 participants and provided demographic information indicating their shift work patterns (regular morning, evening, night, or switching between shifts).

They also completed a questionnaire screening on six common sleep disorder categories: insomnia, hypersomnia, parasomnias, sleep-related breathing disorders, sleep-related movement disorders, and circadian rhythm sleep-wake disorders.

Responses suggested that regular night shifts are the most debilitating condition when it comes to sleep. Half of night shift workers reported sleeping less than 6 hours in a 24-hour period, 51% reported one sleep disorder, and 26% reported two or more sleep disorders.

In the overall study population, approximately one-third tested positive for at least one sleep disorder and 12.6% tested positive for two or more sleep disorders.

Demographic factors and sleep health

Researchers also investigated whether demographic factors such as gender, age, and highest level of education influenced sleep health. We also considered whether participants lived alone, with a partner or children, or with others, such as friends or parents.

The results showed that although men slept less than women, sleep problems were more common in women. Age also affected sleep health. Although older participants tended to sleep less, most sleep disorders and their comorbidities were found to be more prevalent in the youngest participant group, those under 30 years of age.

Researchers found a correlation between education level and the likelihood of having disrupted sleep. “The effects of shift work on sleep are most pronounced among young people with low levels of education,” Lancel said. This group had shorter sleep duration and significantly higher prevalence of sleep disorders and their comorbidities.

Night shifts and sleep challenges

Researchers found that some people who work night shifts may have fewer sleep-related problems than others, but for the average night shift worker, this irregular work pattern can lead to less regular sleep-related problems. They said they would be more likely to struggle with healthy sleep. sleep. “People who work night shifts are unlikely to be completely immune to all the negative effects of night shifts, as they remain focused on their day jobs and out of sync with the environment in which they live,” Lancell said. explained.

The researchers also noted that their study had certain limitations. For example, people with sleep disorders may be more likely to participate in studies focused on sleep than people who sleep well. Nevertheless, the authors said their findings may provide important information for employers in occupations where shift work is common. It may also be used to educate strategies on how to best address and reduce the effects of night work and sleep days.

References: “Shift work is associated with widespread sleep disturbances, especially when working at night,” GJ Boersma, T. Mijnster, P. Vantyghem, GA Kerkhof, Marike Lancel, October 17, 2023. frontiers of psychiatry.
DOI: 10.3389/fpsyt.2023.1233640

Source: scitechdaily.com